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Mitochondrial dysfunction is a key determinant of the rare disease lymphangioleiomyomatosis and provides a novel therapeutic target

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dc.contributor.authorAbdelwahab, E. M. M.
dc.contributor.authorPal, S.
dc.contributor.authorKvell, K.
dc.contributor.authorSarosi, V.
dc.contributor.authorBai, P.
dc.contributor.authorRue, R.
dc.contributor.authorKrymskaya, V.
dc.contributor.authorMcPhail, D.
dc.contributor.authorPorter, A.
dc.contributor.authorPongracz, J. E.
dc.contributor.institutionUniversity of Aberdeen.Medical Sciencesen
dc.date.accessioned2019-01-17T09:40:06Z
dc.date.available2019-01-17T09:40:06Z
dc.date.issued2019
dc.descriptionAcknowledgements The authors are grateful to Prof. Dr. Laszlo Seress, Professor Emeritus, Central Electron Microscope Laboratory, University of Pecs, Pecs, Hungary for his invaluable assistance with electron microscopic studies using the Jeol 1200 TEM and Jeol 1400 TEM electron microscopes. Jeol TEM was funded by the GINOP-2.3.3-15-2016-0002 (New generation electron microscope: 3D ultrastructure). We would also like to thank Dr. Veronika Csongei, PhD, Senior Lecturer, Department of Pharmaceutical Biotechnology and Janos Szentagothai Research Centre, University of Pecs, Pecs, Hungary for assistance with statistical analysis. Funding JEP was supported by the European Union and the State of Hungary, co-financed by the European Social Fund in the framework of TÁMOP-4.2.4.A/2-11/1-2012-0001 “National Excellence Program”.en
dc.description.statusPeer revieweden
dc.format.extent9
dc.format.extent2816832
dc.identifier141706801
dc.identifier6f9d8d96-fbaa-4027-b5bd-c967caf267e9
dc.identifier85058854414
dc.identifier000465167600015
dc.identifier.citationAbdelwahab, E M M, Pal, S, Kvell, K, Sarosi, V, Bai, P, Rue, R, Krymskaya, V, McPhail, D, Porter, A & Pongracz, J E 2019, 'Mitochondrial dysfunction is a key determinant of the rare disease lymphangioleiomyomatosis and provides a novel therapeutic target', Oncogene, vol. 38, pp. 3093-3101. https://doi.org/10.1038/s41388-018-0625-1en
dc.identifier.doi10.1038/s41388-018-0625-1
dc.identifier.issn1476-5594
dc.identifier.otherRIS: urn:4DD4F61B3ED263D6AB099AEA3231E6DD
dc.identifier.otherRIS: Abdelwahab2018
dc.identifier.otherMendeley: 36e6dfde-b7b9-3d37-854b-7497f4cc948b
dc.identifier.urihttp://hdl.handle.net/2164/11805
dc.identifier.urlhttp://www.scopus.com/inward/record.url?scp=85058854414&partnerID=8YFLogxKen
dc.identifier.urlhttp://www.mendeley.com/research/mitochondrial-dysfunction-key-determinant-rare-disease-lymphangioleiomyomatosis-provides-novel-theraen
dc.identifier.vol38en
dc.language.isoeng
dc.relation.ispartofOncogeneen
dc.subjectSDG 3 - Good Health and Well-beingen
dc.subjectBIOGENESISen
dc.subjectCELLen
dc.subjectCHAINen
dc.subjectCYTOCHROME-Cen
dc.subjectESTROGENen
dc.subjectFEATURESen
dc.subjectGENEen
dc.subjectPHOSPHORYLATIONen
dc.subjectTSC2en
dc.subjectQH301 Biologyen
dc.subjectGeneticsen
dc.subjectMolecular Biologyen
dc.subjectCancer Researchen
dc.subjectSupplementary Dataen
dc.subject.lccQH301en
dc.titleMitochondrial dysfunction is a key determinant of the rare disease lymphangioleiomyomatosis and provides a novel therapeutic targeten
dc.typeJournal articleen

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