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miR-29 is an important driver of aging-related phenotypes

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dc.contributor.authorSwahari, Vijay
dc.contributor.authorNakamura, Ayumi
dc.contributor.authorHollville, Emilie
dc.contributor.authorHung, Yu-Han
dc.contributor.authorKanke, Matt
dc.contributor.authorKurtz, C. Lisa
dc.contributor.authorCaravia, Xurde M.
dc.contributor.authorRoiz-Valle, David
dc.contributor.authorHe, Shenghui
dc.contributor.authorKrishnamurthy, Janakiraman
dc.contributor.authorKapoor, Sahil
dc.contributor.authorPrasad, Varun
dc.contributor.authorFlowers, Cornelius
dc.contributor.authorBeck, Matthew V.
dc.contributor.authorBaran-Gale, Jeanette
dc.contributor.authorSharpless, Norman
dc.contributor.authorLópez-Otín, Carlos
dc.contributor.authorSethupathy, Praveen
dc.contributor.authorDeshmukh, Mohanish
dc.contributor.institutionUniversity of Aberdeen.Neuroscienceen
dc.contributor.institutionUniversity of Aberdeen.Institute of Medical Sciencesen
dc.contributor.institutionUniversity of Aberdeen.Medical Sciencesen
dc.date.accessioned2024-09-17T10:30:06Z
dc.date.available2024-09-17T10:30:06Z
dc.date.issued2024-08-27
dc.descriptionThe authors thank Dr. Peter J Havel (UC Davis) for sharing monkey tissue samples. We also thank Dr. Greg Hannon for sending us the mouse embryonic stem cells overexpressing miR‐29b that we used to generate the miR-29TG mice. We appreciate the help of Mervi Eeva, Ying Li, and Bentley Midkiff at the UNC Translational Pathology Laboratory (TPL) for expert technical assistance and Dr. Dale Cowley for assistance in gen- erating the miR-29 transgenic mice. We also thank Janice Weaver and Carolyn Suitt at the UNC Animal Histopathology and the Center for Gas- trointestinal Biology and Disease (CGIBD), respectively, as well as Dr. Andrew Troy for assistance in isolating muscle tissue.en
dc.description.statusPeer revieweden
dc.format.extent11
dc.format.extent1821746
dc.identifier293433748
dc.identifier3dc9e329-feab-4dc5-925f-d4597c15e6db
dc.identifier85202147738
dc.identifier.citationSwahari, V, Nakamura, A, Hollville, E, Hung, Y-H, Kanke, M, Kurtz, C L, Caravia, X M, Roiz-Valle, D, He, S, Krishnamurthy, J, Kapoor, S, Prasad, V, Flowers, C, Beck, M V, Baran-Gale, J, Sharpless, N, López-Otín, C, Sethupathy, P & Deshmukh, M 2024, 'miR-29 is an important driver of aging-related phenotypes', Communications Biology, vol. 7, 1055 . https://doi.org/10.1101/2022.11.29.518429, https://doi.org/10.1038/s42003-024-06735-zen
dc.identifier.doi10.1101/2022.11.29.518429
dc.identifier.issn2399-3642
dc.identifier.otherORCID: /0000-0001-5711-8542/work/168098519
dc.identifier.urihttps://hdl.handle.net/2164/24236
dc.identifier.vol7en
dc.language.isoeng
dc.relation.ispartofCommunications Biologyen
dc.subjectSubstantive connection via eligible employment contracten
dc.subjectageingen
dc.subjectmiRNAsen
dc.subjectR Medicine (General)en
dc.subjectSupplementary Informationen
dc.subjecthttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM1_ESM.pdfen
dc.subjecthttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM2_ESM.pdfen
dc.subjecthttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM5_ESM.pdfen
dc.subjectSupplementary Dataen
dc.subjecthttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM3_ESM.xlsxen
dc.subjecthttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM4_ESM.xlsxen
dc.subject.lccR1en
dc.titlemiR-29 is an important driver of aging-related phenotypesen
dc.typeJournal articleen

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