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Calcineurin signaling promotes Takotsubo syndrome

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dc.contributor.authorBruns, Bastian
dc.contributor.authorAntoniou, Marilena
dc.contributor.authorBaier, Irena
dc.contributor.authorJoos, Maximilian
dc.contributor.authorSevinchan, Meryem
dc.contributor.authorMoog, Marie-Christine
dc.contributor.authorDieterich, Christoph
dc.contributor.authorFriederich, Hans-Christoph
dc.contributor.authorKhan, Hilal
dc.contributor.authorWilson, Heather
dc.contributor.authorHerzog, Wolfgang
dc.contributor.authorDawson, Dana
dc.contributor.authorFrey, Norbert
dc.contributor.authorSchultz, Jobst-Hendrik
dc.contributor.authorBacks, Johannes
dc.contributor.institutionUniversity of Aberdeen.Medical Sciences - Cardiovascular Groupen
dc.contributor.institutionUniversity of Aberdeen.Institute of Medical Sciencesen
dc.contributor.institutionUniversity of Aberdeen.Applied Medicineen
dc.date.accessioned2023-07-17T15:56:01Z
dc.date.available2023-07-17T15:56:01Z
dc.date.issued2023-07
dc.descriptionAcknowledgements We thank P. Nawroth (Department of Endocrinology, University Hospital Heidelberg, Germany) for the opportunity to conduct RIA (corticosterone), HPLC (catecholamines) and automated Cobas (hs-TnT) analysis in his laboratory. S. Martinache (Department of General Internal Medicine and Psychosomatics, University Hospital Heidelberg, Germany), J. Krebs-Haupenthal, S. Harrack and M. Oestringer (all affiliated with the Institute of Experimental Cardiology, Medical Faculty, Heidelberg University, Germany) provided excellent technical assistance. This work was supported by grants from the Deutsche Forschungsgemeinschaft (BA 2258/9-1 and the CRC 1550, INST 35/1699-1) and the Deutsches Zentrum für Herz-Kreislauf-Forschung (DZHK; German Centre for Cardiovascular Research), from the BMBF (German Ministry of Education and Research) to J.B., from the German Cardiac Society (DGK) to B.B., I.B. and M.S., and from the German Heart Foundation (DHS) to M.A. C.D. and N.F. were also supported by the CRC 1550 and DZHK. The funders had no role in study design, data collection and analysis, the decision to publish or preparation of the manuscript.en
dc.description.statusPeer revieweden
dc.format.extent11
dc.format.extent9476462
dc.format.extent4403849
dc.identifier251391437
dc.identifier9b999d3e-73e4-4870-91fa-8a4c4f5de81f
dc.identifier85164746922
dc.identifier.citationBruns, B, Antoniou, M, Baier, I, Joos, M, Sevinchan, M, Moog, M-C, Dieterich, C, Friederich, H-C, Khan, H, Wilson, H, Herzog, W, Dawson, D, Frey, N, Schultz, J-H & Backs, J 2023, 'Calcineurin signaling promotes Takotsubo syndrome', Nature Cardiovascular Research , vol. 2, no. 7, pp. 645-655. https://doi.org/10.1038/s44161-023-00296-wen
dc.identifier.doi10.1038/s44161-023-00296-w
dc.identifier.iss7en
dc.identifier.otherORCID: /0000-0002-1407-7274/work/138945438
dc.identifier.urihttps://hdl.handle.net/2164/21237
dc.identifier.urlhttp://www.scopus.com/inward/record.url?scp=85164746922&partnerID=8YFLogxKen
dc.identifier.vol2en
dc.language.isoeng
dc.relation.ispartofNature Cardiovascular Researchen
dc.subjectSDG 3 - Good Health and Well-beingen
dc.subjectTakotsuboen
dc.subjectstressen
dc.subjectcalcineurinen
dc.subjectheart failureen
dc.subjectepinephrineen
dc.subjectsexen
dc.subjectRZ Other systems of medicineen
dc.subjectSupplementary Dataen
dc.subject.lccRZen
dc.titleCalcineurin signaling promotes Takotsubo syndromeen
dc.typeJournal articleen

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