Swahari, VijayNakamura, AyumiHollville, EmilieHung, Yu-HanKanke, MattKurtz, C. LisaCaravia, Xurde M.Roiz-Valle, DavidHe, ShenghuiKrishnamurthy, JanakiramanKapoor, SahilPrasad, VarunFlowers, CorneliusBeck, Matthew V.Baran-Gale, JeanetteSharpless, NormanLópez-Otín, CarlosSethupathy, PraveenDeshmukh, Mohanish2024-09-172024-09-172024-08-27Swahari, V, Nakamura, A, Hollville, E, Hung, Y-H, Kanke, M, Kurtz, C L, Caravia, X M, Roiz-Valle, D, He, S, Krishnamurthy, J, Kapoor, S, Prasad, V, Flowers, C, Beck, M V, Baran-Gale, J, Sharpless, N, López-Otín, C, Sethupathy, P & Deshmukh, M 2024, 'miR-29 is an important driver of aging-related phenotypes', Communications Biology, vol. 7, 1055 . https://doi.org/10.1101/2022.11.29.518429, https://doi.org/10.1038/s42003-024-06735-z2399-3642ORCID: /0000-0001-5711-8542/work/168098519https://hdl.handle.net/2164/24236The authors thank Dr. Peter J Havel (UC Davis) for sharing monkey tissue samples. We also thank Dr. Greg Hannon for sending us the mouse embryonic stem cells overexpressing miR‐29b that we used to generate the miR-29TG mice. We appreciate the help of Mervi Eeva, Ying Li, and Bentley Midkiff at the UNC Translational Pathology Laboratory (TPL) for expert technical assistance and Dr. Dale Cowley for assistance in gen- erating the miR-29 transgenic mice. We also thank Janice Weaver and Carolyn Suitt at the UNC Animal Histopathology and the Center for Gas- trointestinal Biology and Disease (CGIBD), respectively, as well as Dr. Andrew Troy for assistance in isolating muscle tissue.111821746engSubstantive connection via eligible employment contractageingmiRNAsR Medicine (General)Supplementary Informationhttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM1_ESM.pdfhttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM2_ESM.pdfhttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM5_ESM.pdfSupplementary Datahttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM3_ESM.xlsxhttps://static-content.springer.com/esm/art%3A10.1038%2Fs42003-024-06735-z/MediaObjects/42003_2024_6735_MOESM4_ESM.xlsxR1miR-29 is an important driver of aging-related phenotypesJournal article10.1101/2022.11.29.5184297