Šoltić, DarijaShorrock, Hannah K.Allardyce, HazelWilson, Emma L.Holt, IanSynowsky, Silvia A.Shirran, Sally L.Parson, Simon H.Gillingwater, Thomas H.Fuller, Heidi R.2020-08-082020-08-082019-11-01Šoltić, D, Shorrock, H K, Allardyce, H, Wilson, E L, Holt, I, Synowsky, S A, Shirran, S L, Parson, S H, Gillingwater, T H & Fuller, H R 2019, 'Lamin A/C dysregulation contributes to cardiac pathology in a mouse model of severe spinal muscular atrophy', Human Molecular Genetics, vol. 28, no. 21, pp. 3515–3527. https://doi.org/10.1093/hmg/ddz1950964-6906Mendeley: 973731f3-5bd2-354f-9446-bc1938837c77ORCID: /0000-0002-8848-8738/work/82379627https://hdl.handle.net/2164/14887The authors would like to thank Prof Colin L Stewart, Institute of Medical Biology, Singapore, for kindly providing wild type and LMNA knockout mouse embryonic fibroblasts, and Prof Glenn E Morris for helpful discussions about lamin A and for providing access to laboratory equipment. This research was supported by funding from the Newlife Charity [SG/15-16/11] (HF) and Keele University ACORN funding (HF & DS); British Heart Foundation [PG/16/68/31991] (IH); UK SMA Research Consortium (SMA Trust) (THG) and the Euan MacDonald Centre for Motor Neurone Disease Research (HKS and THG); and Wellcome Trust [094476/Z/10/Z] (SLS).132460373engR Medicine (General)L Education (General)OtherSG/15-16/11British Heart FoundationPG/16/68/31991Wellcome Trust094476/Z/10/ZSupplementary DataR1L1Journal article10.1093/hmg/ddz195http://www.mendeley.com/research/lamin-ac-dysregulation-contributes-cardiac-pathology-mouse-model-severe-spinal-muscular-atrophy2821