Development of abnormalities at the neuromuscular junction in the SOD1-G93A mouse model of ALS : dysfunction then disruption of postsynaptic structure precede overt motor symptoms

McIntosh, JayneMekrouda, ImaneDashti, MaryamGiuraniuc, Claudiu V.Banks, Robert W.Miles, Gareth B.Bewick, Guy S.2023-10-182023-10-182023-05-19McIntosh, J, Mekrouda, I, Dashti, M, Giuraniuc, C V, Banks, R W, Miles, G B & Bewick, G S 2023, 'Development of abnormalities at the neuromuscular junction in the SOD1-G93A mouse model of ALS : dysfunction then disruption of postsynaptic structure precede overt motor symptoms', Frontiers in Molecular Neuroscience, vol. 16, 1169075. https://doi.org/10.3389/fnmol.2023.11690751662-5099Bibtex: 10.3389/fnmol.2023.1169075Bibtex: 10.3389/fnmol.2023.1169075ORCID: /0000-0001-8266-7797/work/145600620https://hdl.handle.net/2164/21949Funding This project was funded by a PhD studentship to JM from Motor Neurone Disease Scotland, on grant number RGB3753 to GB and GM.166495017engneuromuscular junctionmotor neuron diseaseneuromuscular transmissionSOD1-G93AALSacetylcholine receptormouseR MedicineOtherRGB3753RDevelopment of abnormalities at the neuromuscular junction in the SOD1-G93A mouse model of ALS : dysfunction then disruption of postsynaptic structure precede overt motor symptomsJournal article10.3389/fnmol.2023.116907516