Garfield, Alastair SDavies, Jennifer RBurke, Luke KFurby, Hannah VWilkinson, Lawrence SHeisler, Lora KIsles, Anthony R2017-01-302017-01-302016-12-08Garfield, A S, Davies, J R, Burke, L K, Furby, H V, Wilkinson, L S, Heisler, L K & Isles, A R 2016, 'Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite', Molecular brain, vol. 9, no. 1, 95. https://doi.org/10.1186/s13041-016-0277-41756-6606PubMedCentral: PMC5144496ORCID: /0000-0002-7731-1419/work/30894622http://hdl.handle.net/2164/8078Funding This work was supported by grants from the Prader-Willi Syndrome Association UK, Wellcome Trust (WT098012; WT081713 to LKH), the Foundation for Prader-Willi Research (to ARI), the Medical Research Council (G0801418) and the Biotechnology and Biological Research Council (BB/J016756/1; to LSW and ARI).1097561engSDG 3 - Good Health and Well-beingSnord115Prader-Willi syndromeSerotonin 2C receptorAlternate splicingfeedingRC0321 Neuroscience. Biological psychiatry. NeuropsychiatryWellcome TrustWT098012WT081713Medical Research Council (MRC)G0801418Biotechnology and Biological Sciences Research Council (BBSRC)BB/J016756/1RC0321Journal article10.1186/s13041-016-0277-491