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Early changes in [18F] FDG incorporation by breast cancer cells treated with trastuzumab in normoxic conditions : Role of the Akt-pathway, glucose transport and HIF-1α

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dc.contributor.authorFleming, Ian Neil
dc.contributor.authorAndriu, Alexandra
dc.contributor.authorSmith, Timothy Andrew Davies
dc.contributor.institutionUniversity of Aberdeen.Applied Medicineen
dc.contributor.institutionUniversity of Aberdeen.Aberdeen Biomedical Imaging Centreen
dc.contributor.institutionUniversity of Aberdeen.Medical Sciencesen
dc.date.accessioned2014-11-29T00:01:45Z
dc.date.available2014-11-29T00:01:45Z
dc.date.embargoedUntil2015-04-01
dc.date.issued2014-04-01
dc.descriptionAcknowledgment The work was funded by the University of Aberdeen and Friends of Anchor. We thank Dr Iain Brown, University of Aberdeen, for the generous gift of the anti-pHER2 antibody.en
dc.description.statusPeer revieweden
dc.format.extent8
dc.format.extent1571368
dc.format.extent1571368
dc.identifier32495059
dc.identifier4dc2688f-8225-40d8-afe2-28143d76f6d5
dc.identifier84896544782
dc.identifier.citationFleming, I N, Andriu, A & Smith, T A D 2014, 'Early changes in [18F] FDG incorporation by breast cancer cells treated with trastuzumab in normoxic conditions : Role of the Akt-pathway, glucose transport and HIF-1α', Breast Cancer Research and Treatment, vol. 144, no. 4, pp. 241-248. https://doi.org/10.1007/s10549-014-2858-1en
dc.identifier.doi10.1007/s10549-014-2858-1
dc.identifier.iss4en
dc.identifier.issn0167-6806
dc.identifier.otherORCID: /0000-0001-9505-6418/work/86450261
dc.identifier.urihttp://hdl.handle.net/2164/4034
dc.identifier.vol144en
dc.language.isoeng
dc.relation.ispartofBreast Cancer Research and Treatmenten
dc.subjectSDG 3 - Good Health and Well-beingen
dc.subject[18F]FDGen
dc.subjectglucose transporten
dc.subjectPI3/AKTen
dc.subjecttrastuzumaben
dc.subjectRC0254 Neoplasms. Tumors. Oncology (including Cancer)en
dc.subject.lccRC0254en
dc.titleEarly changes in [18F] FDG incorporation by breast cancer cells treated with trastuzumab in normoxic conditions : Role of the Akt-pathway, glucose transport and HIF-1αen
dc.typeJournal articleen

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