Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite

Garfield, Alastair S; Davies, Jennifer R; Burke, Luke K; Furby, Hannah V; Wilkinson, Lawrence S; Heisler, Lora K; Isles, Anthony R

Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite

Files

Increased_alternate_splicing_of_Htr2c_in_a_mouse_model_for_Prader_Willi_syndrome_leads_disruption_of_5HT2C_receptor_mediated_appetite.pdf (1.05 MB)

Date

2016-12-08

Authors

Wilkinson, Lawrence S

Heisler, Lora K

Isles, Anthony R

Citation

Garfield, A S, Davies, J R, Burke, L K, Furby, H V, Wilkinson, L S, Heisler, L K & Isles, A R 2016, 'Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite', Molecular brain, vol. 9, no. 1, 95. https://doi.org/10.1186/s13041-016-0277-4