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Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite

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dc.contributor.authorGarfield, Alastair S
dc.contributor.authorDavies, Jennifer R
dc.contributor.authorBurke, Luke K
dc.contributor.authorFurby, Hannah V
dc.contributor.authorWilkinson, Lawrence S
dc.contributor.authorHeisler, Lora K
dc.contributor.authorIsles, Anthony R
dc.contributor.institutionUniversity of Aberdeen.Rowett Instituteen
dc.date.accessioned2017-01-30T09:28:18Z
dc.date.available2017-01-30T09:28:18Z
dc.date.issued2016-12-08
dc.descriptionFunding This work was supported by grants from the Prader-Willi Syndrome Association UK, Wellcome Trust (WT098012; WT081713 to LKH), the Foundation for Prader-Willi Research (to ARI), the Medical Research Council (G0801418) and the Biotechnology and Biological Research Council (BB/J016756/1; to LSW and ARI).en
dc.description.statusPeer revieweden
dc.format.extent1097561
dc.identifier85409673
dc.identifierd6dd7b0c-ef27-4bc2-aa74-331eddbc6095
dc.identifier27931246
dc.identifier85002528943
dc.identifier.citationGarfield, A S, Davies, J R, Burke, L K, Furby, H V, Wilkinson, L S, Heisler, L K & Isles, A R 2016, 'Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite', Molecular brain, vol. 9, no. 1, 95. https://doi.org/10.1186/s13041-016-0277-4en
dc.identifier.doi10.1186/s13041-016-0277-4
dc.identifier.iss1en
dc.identifier.issn1756-6606
dc.identifier.otherPubMedCentral: PMC5144496
dc.identifier.otherORCID: /0000-0002-7731-1419/work/30894622
dc.identifier.urihttp://hdl.handle.net/2164/8078
dc.identifier.vol9en
dc.language.isoeng
dc.relation.ispartofMolecular brainen
dc.subjectSDG 3 - Good Health and Well-beingen
dc.subjectSnord115en
dc.subjectPrader-Willi syndromeen
dc.subjectSerotonin 2C receptoren
dc.subjectAlternate splicingen
dc.subjectfeedingen
dc.subjectRC0321 Neuroscience. Biological psychiatry. Neuropsychiatryen
dc.subjectWellcome Trusten
dc.subjectWT098012en
dc.subjectWT081713en
dc.subjectMedical Research Council (MRC)en
dc.subjectG0801418en
dc.subjectBiotechnology and Biological Sciences Research Council (BBSRC)en
dc.subjectBB/J016756/1en
dc.subject.lccRC0321en
dc.titleIncreased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetiteen
dc.typeJournal articleen

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